搜索结果: 'stemprep kits'

1:44

Video
Elevate Tissue Dissociation with STEMprep™

发布日期: 4/29/25
人IgE ELISA Kit 用于人免疫球蛋白E的检测和测定
Tech Tip
Optimization of EasySep™ Releasable RapidSpheres™ Indirect Positive Selection Kits
Brochure
ELISA Kits for Immunology Research

产品类型：

ELISAs

品牌：

ELISA

产品号#：

02000

02001

02002

02003

02006

02007

02008

02009

02010

02011

02012

02013

02014

02015

02016

02017

02018

02019

02020

02021

02022

02023

02024

02025

02026

02027

02028

02029

02030

02031

01916

01917

01992

01993

01994

01995

01996

01997

01998

01999

02004

02005

020

产品名：

人ifn - α酶联免疫吸附测定试剂盒

人ifn - α酶联免疫吸附测定试剂盒

人ifn - γ酶联免疫吸附测定试剂盒

人ifn - γ酶联免疫吸附测定试剂盒

人IL-2酶联免疫吸附测定试剂盒

人IL-2酶联免疫吸附测定试剂盒

人IL-4酶联免疫吸附测定试剂盒

人IL-4酶联免疫吸附测定试剂盒

人IL-5酶联免疫吸附测定试剂盒

人IL-5酶联免疫吸附测定试剂盒

人IL-10酶联免疫吸附测定试剂盒

人IL-10酶联免疫吸附测定试剂盒

人IL-12 （p70）酶联免疫吸附测定试剂盒

人IL-12 （p70）酶联免疫吸附测定试剂盒

人IL-23酶联免疫吸附测定试剂盒

人IL-23酶联免疫吸附测定试剂盒

人潜伏tgf - β 1酶联免疫吸附测定试剂盒

小鼠ifn - γ酶联免疫吸附测定试剂盒

小鼠ifn - γ酶联免疫吸附测定试剂盒

小鼠IL-2酶联免疫吸附测定试剂盒

小鼠IL-2酶联免疫吸附测定试剂盒

小鼠IL-5酶联免疫吸附测定试剂盒

小鼠IL-5酶联免疫吸附测定试剂盒

小鼠IL-12 （p70）酶联免疫吸附测定试剂盒

小鼠IL-12 （p70）酶联免疫吸附测定试剂盒

小鼠IL-12/-23 （p40）酶联免疫吸附测定试剂盒

小鼠tnf - α酶联免疫吸附测定试剂盒

小鼠tnf - α酶联免疫吸附测定试剂盒

ELISA稀释剂

pNPP ELISA试剂盒

人IgA ELISA抗体对试剂盒

人IgE ELISA抗体对试剂盒

人IgG ELISA抗体对试剂盒

人IgM抗体对ELISA试剂盒

小鼠IgA ELISA抗体对试剂盒

小鼠IgE ELISA抗体对试剂盒

小鼠IgG ELISA抗体对试剂盒

小鼠IgM抗体对ELISA试剂盒

人IL-1 β酶联免疫吸附测定试剂盒

人IL-1 β酶联免疫吸附测定试剂盒
EasySep™人CD45去除试剂盒II 免疫磁珠法去除人CD45+细胞
Brochure
Elevate Tissue Dissociation with STEMprep™

产品类型：

Instruments and Software

品牌：

STEMprep

产品号#：

产品名：
EasySep™ Human ILC2 Enrichment Kit 免疫磁珠负选细胞富集试剂盒
Technical Bulletin
Isolating Extracellular Vesicles from Urine with EasySep™ EV Human Positive Selection Kits

产品类型：

Cell Isolation Products

细胞类型：

其它细胞系

产品号#：

产品名：
Brochure
EasySep™ Direct Human PBMC Isolation Kit

产品类型：

Cell Isolation Products

品牌：

EasySep，RoboSep

产品号#：

19654

19654RF

产品名：

EasySep™直接人PBMC分离试剂盒

EasySep™直接人PBMC分离试剂盒
Reference

Kim M-H et al. (MAR 2011) Blood 117 12 3343--52

Neutrophil survival and c-kit(+)-progenitor proliferation in Staphylococcus aureus-infected skin wounds promote resolution.

Polymorphonuclear neutrophils (PMNs) are critical for the formation,maintenance,and resolution of bacterial abscesses. However,the mechanisms that regulate PMN survival and proliferation during the evolution of an abscess are not well defined. Using a mouse model of Staphylococcus aureus abscess formation within a cutaneous wound,combined with real-time imaging of genetically tagged PMNs,we observed that a high bacterial burden elicited a sustained mobilization of PMNs from the bone marrow to the infected wound,where their lifespan was markedly extended. A continuous rise in wound PMN number,which was not accounted for by trafficking from the bone marrow or by prolonged survival,was correlated with the homing of c-kit(+)-progenitor cells from the blood to the wound,where they proliferated and formed mature PMNs. Furthermore,by blocking their recruitment with an antibody to c-kit,which severely limited the proliferation of mature PMNs in the wound and shortened mouse survival,we confirmed that progenitor cells are not only important contributors to PMN expansion in the wound,but are also functionally important for immune protection. We conclude that the abscess environment provides a niche capable of regulating PMN survival and local proliferation of bone marrow-derived c-kit(+)-progenitor cells. View Publication

产品类型：

产品号#：

03434

03444

产品名：

MethoCult™GF M3434

MethoCult™GF M3434
EasySep™小鼠T细胞分选试剂盒 不带标记的小鼠T细胞免疫磁珠负选
Reference

Zhang P et al. (FEB 2009) Journal of immunology (Baltimore,Md. : 1950) 182 3 1568--76

Acute alcohol intoxication inhibits the lineage- c-kit+ Sca-1+ cell response to Escherichia coli bacteremia.

Alcohol abuse predisposes the host to bacterial infections. In response to bacterial infection,the bone marrow hematopoietic activity shifts toward granulocyte production,which is critical for enhancing host defense. This study investigated the hematopoietic precursor cell response to bacteremia and how alcohol affects this response. Acute alcohol intoxication was induced in BALB/c mice 30 min before initiation of Escherichia coli bacteremia. Bacteremia caused a significant increase in the number of bone marrow lineage (lin(-))-c-kit(+)Sca-1(+) cells. Marrow lin(-)c-kit(+)Sca-1(+) cells isolated from bacteremic mice showed an increase in CFU-granulocyte/macrophage activity compared with controls. In addition to enhanced proliferation of lin(-)c-kit(+)Sca-1(+) cells as reflected by BrdU incorporation,phenotypic inversion of lin(-)c-kit(+)Sca-1(+)Sca-1(-) cells primarily accounted for the rapid increase in marrow lin(-)c-kit(+)Sca-1(+) cells following bacteremia. Bacteremia increased plasma concentration of TNF-alpha. Culture of marrow lin(-)c-kit(+)Sca-1(+)Sca-1(-) cells with murine rTNF-alpha for 24 h caused a dose-dependent increase in conversion of these cells to lin(-)c-kit(+)Sca-1(+) cells. Sca-1 mRNA expression by the cultured cells was also up-regulated following TNF-alpha stimulation. Acute alcohol intoxication inhibited the increase in the number of lin(-)c-kit(+)Sca-1(+) cells in the bone marrow after E. coli infection. Alcohol impeded the increase in BrdU incorporation into marrow lin(-)c-kit(+)Sca-1(+) cells in response to bacteremia. Alcohol also suppressed the plasma TNF-alpha response to bacteremia and inhibited TNF-alpha-induced phenotypic inversion of lin(-)c-kit(+)Sca-1(+)Sca-1(-) cells in vitro. These data show that alcohol inhibits the hematopoietic precursor cell response to bacteremia,which may serve as one mechanism underlying the impaired host defense in alcohol abusers with severe bacterial infections. View Publication

产品类型：

产品号#：

03534

03434

03444

09600

09650

产品名：

MethoCult™GF M3534

MethoCult™GF M3434

MethoCult™GF M3434

StemSpan™SFEM

StemSpan™SFEM